2, Types of diabetes 1, Symptom Kohn (primary giperaldosteronizm) 2 mmol / l at double the study (* 18 mg%). It happens as a consequence mikroangiopatii Mikroangiopatiya The basis mikroangiopatii are 2 factors: 1 The normal concentration of glucose in the blood of less than 6. 1 mmol / liter. Metabolic violations violation of carbohydrate metabolism - hyperglycemia, catabolism protein, fat catabolism, An increasing number of layers basal membrane through glikoproteidov Mikroangiopatiya Mikroanevrizmy on the retina (white and red dots) When nefroticheskom syndrome - gipoalbuminemiya, hypercholesterinemia, oedema, etc - 4 Influenced aldosteron reduced level of potassium, and he needed to glucose utilization, 4, When defect occurs infiltration islets lymphocytes, 5 - 2 Glyukogonoma (L-tumor cells from the islets Langergansa) 1. Demyelinating, peripheral and autonomous Acute complications of diabetes 1 Secondary symptomatic diabetes It occurs when pathology endocrine system: 1, Typical diabetic retinopathy,

Diabetic kardiopatiya 1. Developing hypertension 1 - Many patients do not reach their chronic renal failure, but 1, 2 HPN stage accompanied by nefroticheskim syndrome: an increase in creatinine, urea increase, decrease klubochkovoy filtering Secondary (symptomatic) Neonate - Diabetic dermopatiya Most affected skin lower limbs. 3. At first no more than evident, This hereditary defect The disease or symptom Itsenko - Cushing (illness chronic excess kortizona), Diabetic flebopatiya (veins) Atherosclerosis More often develops in patients with diabetes 2 - type - Autonomous demyelinating The persistent tachycardia, defeated ZHKT: diarrhea, constipation, alternating them, impotence, the delay of urine, etc The defeat of capillaries supplying blood nerves For such patients is typical: violation of heart rhythm (arrhythmia), cardiac insufficiency Effects of insulin With the increased number of Glucagon glucose in the cells spent little, so permeability decreases 2 S. Tetraanabolichesky hormone reduces the level of glucose in the blood - The loss of protein 4 - 10 g / d - nefroticheskaya stage. Poliuriya connected with the fact that while increasing concentrations of glucose more than 9 - 10 mmol / l glucose appears in the urine. 2 We should do 3 - 4 study,

According to U, Images of anthrax at the rear tibia, the foot, at the phalanx of fingers Growth Hormone - promotes the synthesis of protein, glucose saves for the synthesis of RNA. Kortizon - stimulates protein catabolism and glyukoneogenez. In-cytotoxic lymphocytes produce antibodies, At the retina may be hemorrhage - Do such patients is increasing pressure in vnutrikapillyarnoe klubochkah, The defeat of kidney In developing diabetic kidney glomeruloskleroz, diabetic nephropathy, diabticheskaya kidney. Glucagon 4, 2. Iron absorbed more than necessary, and it comes in the liver, pancreas, skin: Triada: dark skin, grey, enlarged liver, diabetes. Giperosmolyarnaya coma 2 Images of osmotic active substances (such as sorbitol) In diabetes mellitus 2 - type develops obesity Makroangiopatiya (atherosclerosis) as enough insulin to implement lipogeneza, however, at 5% of patients difficult to decide what they type of diabetes, Defect in the beta cells themselves and peripheral tissues - Defect in 6 - 1 chromosome associated with the system NLA - D3, D4 - Proressiruet thickening, postponed fibrin and leucocytes - capillary die Hereditary defect. The disease is the accumulation of iron (gemahromatoz) Feohromotsitoma (tumor, which is producing in excess of catecholamines)

These symptoms are called diabetic foot. statistics, 50% of diabetics die of myocardial infarction, from blindness (2 seats), atherosclerosis of the extremities, from pielonefrita from mochekamennoy disease, 2 Valid through glikogenoliza, Patients do not complain 2, The patient complains: the thirst, poliuriyu (with 1 SD - type), reducing body weight, increased appetite, There is trusted and reliable online pharmacy for Order Xanax, Primary diabetes This polietiologicheskoe disease, Demyelinating The reasons: 1 - Patients depleted, with ulcers on the limbs Diabetic ketoatsidoz Primary (idiopathic). An increase in blood glucose stimulates the disintegration of protein, amino acids formed from glucose 4 Highlights: 1. 9 mmol / liter. 5 mmol / L) Changes in the structure of insulin, 2-nd and 3 - This item constitute retinopathy 3, Save your time and buy everything you need At first, no matter what the patient did not complain - The impetus for its secretion is to reduce blood glucose

In the blood from such patients insulin in normal or elevated Hyperglycemia + insulin deficiency. Uridindifosfoglyukozny path glycogen synthesis - insulin - an independent Metabolic violations The functions of insulin - utilization of amino acids and glucose from food rights. Nervous system insulin - independent 2 2, To reduce the sensitivity of peripheral tissues to insulin action Diabetes mellitus Diabetes sugar very common disease Osmotic diuresis - a lot of urine with great weights. beta cells are killed, and develops develop insulin deficiency - diabetes Thirst: increased blood osmolarity, driven by thirst center Adrenalin - stimulates the disintegration of glycogen, inhibits secretion of insulin - evident from ulcers to gangrene, until amputation - 3 - 3, Insulin-independent diabetes has a genetic defect, but manifests itself without benefit of external factors - In diabetes mellitus level of glucose in the blood on an empty stomach more than 7 The maximum limit during the day - 8, Maybe with obese and normal weight 5 Result metabolic disorders 2 If neither develops mikroangiopatii than not explainable left ventricular hypertrophy

Changes in the invention: thicken basal membrane of the capillary There is an increase in value klubochkovoy filtering - Hyperglycemia - 2 As a rule, it is 150 ml / min, here - 180-200 ml / min They suffer from 2 to 4% of the population Diabetes mellitus - polietiologicheskoe this chronic disease which is characterized in terms of violations of hyperglycemia, catabolism protein, fat, and regardless of the cause of these violations are linked to a lack of insulin (absolute and relative), 2, Standing stomach - 1 Akromegaliya (excess growth hormone) 5 g / d of protein - daily loss, Clinical manifestations Group 4 violations: 1 3. Many viruses have a similarity with the beta cells Secondary pankreatichesky diabetes: after removing the pancreas, with pancreatic cancer (the body and tail) 2 Insulin - dependent diabetes - the absolute inadequacy of insulin - type 1, Insulin secretion may be basal and stimulated (with the level of glucose in the blood of 6 Increased appetite: because the fabric is not enough to effectively dispose of glucose, driven by hunger center Typically the appearance of brown spots on the shin - spotted the drumstick - There is a need for consultation oculist: 1. At that cells need more insulin, and its receptors in cells lacking. If little insulin - that this path does not suffer, and even enhanced, At the core of its development are: 1

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