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Changes in the invention: thicken basal membrane of the capillary, Kortizon - stimulates protein catabolism and glyukoneogenez Thirst: increased blood osmolarity, driven by thirst center - Highlights: 1. 2, 3 3, Many viruses have a similarity with the beta cells. 3 beta cells are killed, and develops develop insulin deficiency - diabetes Growth Hormone - promotes the synthesis of protein, glucose saves for the synthesis of RNA The defeat of capillaries supplying blood nerves Mikroangiopatiya. If little insulin - that this path does not suffer, and even enhanced Secondary symptomatic diabetes It occurs when pathology endocrine system: 1 It happens as a consequence mikroangiopatii Nervous system insulin - independent We should do 3 - 4 study. 5 mmol / L) Diabetes mellitus Diabetes sugar very common disease 1 - Standing stomach - 1 - 4 According to U -

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Autonomous demyelinating The persistent tachycardia, defeated ZHKT: diarrhea, constipation, alternating them, impotence, the delay of urine, etc, Reducing body weight: kontrinsulyarnye factors have lipoliticheskim operation -> weight loss - The major with relative insulin deficiency. Akromegaliya (excess growth hormone), Tetraanabolichesky hormone reduces the level of glucose in the blood. 2 Hereditary defect, There is an increase in value klubochkovoy filtering They suffer from 2 to 4% of the population Symptom Kohn (primary giperaldosteronizm) When defect occurs infiltration islets lymphocytes, Such patients: tewi heart tones, systolic noise, the EKG low, flat, izoelektricheskie wave T. Changes in the structure of insulin Hyperglycemia + insulin deficiency Diabetic flebopatiya (veins) - Increased appetite: because the fabric is not enough to effectively dispose of glucose, driven by hunger center Many patients do not reach their chronic renal failure, but 1, 2 HPN stage accompanied by nefroticheskim syndrome: an increase in creatinine, urea increase, decrease klubochkovoy filtering. Uridindifosfoglyukozny path glycogen synthesis - insulin - an independent Insulin - dependent diabetes - the absolute inadequacy of insulin - type 1 In the normal level of iron in the blood is regulated by a feedback mechanism 2 - The maximum limit during the day - 8. He opposed: 1 First, casual (tranzitornaya)

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In diabetes mellitus 2 - type develops obesity Glyukogonoma (L-tumor cells from the islets Langergansa) Developing hypertension. Giperosmolyarnaya coma. 2. The disease is the accumulation of iron (gemahromatoz). 4 - Secondary (symptomatic) Result metabolic disorders. At the core of its development are: 1 4, The patient complains: the thirst, poliuriyu (with 1 SD - type), reducing body weight, increased appetite Atherosclerosis More often develops in patients with diabetes 2 - type Diabetic dermopatiya Most affected skin lower limbs 5 g / d of protein - daily loss 2. Mikroangiopatiya The basis mikroangiopatii are 2 factors: 1 - When nefroticheskom syndrome - gipoalbuminemiya, hypercholesterinemia, oedema, etc. Images of osmotic active substances (such as sorbitol) There is a need for consultation oculist: 1 Insulin is a growth factor, insulin stimulates the proliferation gladkomyshechnyh fibers arteries and stimulates the synthesis of cholesterol, which postponed gladkomyshechnyh cells in the arteries. 2 mmol / l at double the study (* 18 mg%). evident from ulcers to gangrene, until amputation - 5 - 2

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